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One or more keywords matched the following items that are connected to Weichselbaum, Ralph R.
Item TypeName
Concept Proto-Oncogene Proteins c-abl
Academic Article The stress response to ionizing radiation involoves c-Abl-dependent phosphorylation of SHPTP1.
Academic Article Role for c-Abl tyrosine kinase in growth arrest response to DNA damage.
Academic Article Functional interaction between DNA-PK and c-Abl in response to DNA damage.
Academic Article Inhibition of phosphatidylinositol 3-kinase by c-Abl in the genotoxic stress response.
Academic Article Activation of protein kinase C delta by the c-Abl tyrosine kinase in response to ionizing radiation.
Academic Article Regulation of the rapamycin and FKBP-target 1/mammalian target of rapamycin and cap-dependent initiation of translation by the c-Abl protein-tyrosine kinase.
Academic Article Regulation of Rad51 function by c-Abl in response to DNA damage.
Academic Article Determination of cell fate by c-Abl activation in the response to DNA damage.
Academic Article p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage.
Academic Article Functional role for the c-Abl tyrosine kinase in meiosis I.
Academic Article Activation of MEK kinase 1 by the c-Abl protein tyrosine kinase in response to DNA damage.
Academic Article Interaction of hematopoietic progenitor kinase 1 and c-Abl tyrosine kinase in response to genotoxic stress.
Academic Article Activation of the cytoplasmic c-Abl tyrosine kinase by reactive oxygen species.
Academic Article c-Abl tyrosine kinase regulates caspase-9 autocleavage in the apoptotic response to DNA damage.
Academic Article Activation of the c-Abl tyrosine kinase in the stress response to DNA-damaging agents.
Academic Article Activation of p38 mitogen-activated protein kinase by c-Abl-dependent and -independent mechanisms.
Academic Article Regulation of DNA damage-induced apoptosis by the c-Abl tyrosine kinase.
Academic Article Functional role for the c-Abl protein tyrosine kinase in the cellular response to genotoxic stress.
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  • Oncogene Proteins v abl